Search results for "oval cells"

showing 3 items of 3 documents

TCDD deregulates contact inhibition in rat liver oval cells via Ah receptor, JunD and cyclin A.

2007

The aryl hydrocarbon receptor (AhR) is a transcription factor involved in physiological processes, but also mediates most, if not all, toxic responses to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD). Activation of the AhR by TCDD leads to its dimerization with aryl hydrocarbon nuclear translocator (ARNT) and transcriptional activation of several phase I and II metabolizing enzymes. However, this classical signalling pathway so far failed to explain the pleiotropic hazardous effects of TCDD, such as developmental toxicity and tumour promotion. Thus, there is an urgent need to define genetic programmes orchestrated by AhR to unravel its role in physiology and toxicology. Here we show that TCDD …

Cancer ResearchAryl hydrocarbon receptor nuclear translocatorPolychlorinated Dibenzodioxinscyclin AProto-Oncogene Proteins c-junCyclin DCyclin Acell cycle controlCyclin ATetrachlorodibenzodioxinModels BiologicalDownregulation and upregulationGeneticsAnimalsRNA Small InterferingMolecular BiologyTranscription factorAryl hydrocarbon receptorCells CulturedbiologyContact InhibitionContact inhibitionCell cycleAryl hydrocarbon receptorRatsAdult Stem CellsLiverReceptors Aryl Hydrocarbonliver oval cellsbiology.proteinCancer researchJunDOncogene
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Hepatic progenitors for liver disease: current position

2010

Alice Conigliaro1, David A Brenner2, Tatiana Kisseleva21University “La Sapienza”, Dipartimento di Biotecnologie Cellulari ed Ematologia Policlinico Umberto I, V Clinica Medica, Rome, Italy; 2Department of Medicine, University of California, San Diego, La Jolla, CA, USAAbstract: Liver regeneration restores the original functionality of hepatocytes and cholangiocytes in response to injury. It is regulated on several levels, with different cellular populations contributing to this process, eg, hepatocytes, liver precursor cells, intrahepatic stem cells. In response to injury, mature hepatocytes have the capability to proliferate and give rise to new hepatocytes and cholangi…

Pathologymedicine.medical_specialtyoval cellsLiver cytologyMedicine (miscellaneous)cholangiocytes; hepatic progenitor; hepatocytes; intrahepatic stem cells; liver disease; liver precursor cells; oval cellsReviewBiologyhepatocytemedicinecholangiocytesProgenitor cellliver precursor cellQH573-671Mesenchymal stem cellintrahepatic stem cellCell Biologyhepatic progenitorliver precursor cellsintrahepatic stem cellsLiver regenerationCell biologyHaematopoiesisAmniotic epithelial cellsHepatic stellate cellhepatocytesStem cellCytologyliver diseasecholangiocyteStem Cells and Cloning: Advances and Applications
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Aryl hydrocarbon receptor-dependent cell cycle arrest in isolated mouse oval cells

2013

The aryl hydrocarbon receptor (AhR) is a ligand-activated transcription factor, which mediates toxic responses to environmental pollutants, such as 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) and related compounds. Besides its well known role in induction of xenobiotic metabolizing enzymes, for instance CYP1A1, the AhR is also involved in tumor promotion in rodents although the underlying mechanisms are still poorly understood. Additionally, the AhR is known to regulate cellular proliferation, which might result in either inhibition or stimulation of proliferation depending on the cell-type studied. Potential targets in hepatocarcinogenesis are liver oval (stem/progenitor) cells. In the pres…

medicine.medical_specialtyTCDDPolychlorinated DibenzodioxinsCell cycle checkpointBlotting WesternCyclin AMice TransgenicCyclin ATransfectionToxicologyRetinoblastoma ProteinCell LineMiceCyclin D1Proliferating Cell Nuclear AntigenInternal medicinemedicineAnimalsCyclin D1RNA Small InterferingTranscription factorCell Proliferationbiologyaryl hydrocarbon receptorRetinoblastoma proteinmouse oval cellsCell Cycle CheckpointsGeneral MedicineCell cycleAryl hydrocarbon receptorCell biologyEndocrinologyLiverReceptors Aryl Hydrocarbonbiology.proteinEnvironmental PollutantsTumor promotioncell cycle
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